Researchers extend the lifetime of the qubit.

 

Image 1: Molecular qubit

Researchers extended the lifetime of the molecular qubits. Many things are making molecular qubits interesting tools in quantum computing. 

Researchers can create molecular qubits by putting some atoms (like krypton, helium, gold, etc.) in the fullerene molecule. Then they are driving information to that atom.

Then that atom sends the information to the fullerene core. And after that, the fullerene core will transmit the data to the multiple receiving sensors. If that system uses a C60 molecule. That allows the creation of a qubit that has 60 channels. 

Image 2: C60 Fullerene

Also, receivers have numbers. The system uses numbered data segments. So the data line will delete all other segments than the segment whose number matches the number of the data line. So when data arrives at line 5 the receiver destroys all other data segments than segment number 5. And that allows putting the data row into pieces. Then the system turns the data row into a line. 

The thing that makes qubit so important is that it is required in the process. That transforms the data row into the data line. Diagram 1. There is a possibility to create qubits with four states by using electric wires when the temperature is as low as possible. But the molecule-based qubits are more useful in biological microprocessors. 

Diagram 1: Qubit's principle. 

A =Data row 1

B= The level that transforms data from row to linear form. 

C= Datarow 2. Where the system returns linear data in a row. 

The biological qubits can also transfer data to neurons or microprocessors. Genetic engineering allows researchers can create bacteria or viruses that can operate as a qubit. 

Researchers are the first time manipulated data inside the DNA. And that allows the creation of artificial bacteria which can transfer information to neurons or microprocessors. The idea is that the DNA transfer will make bacteria able to send electric impulses. Then the data that is stored in the DNA will transfer to the receivers which can be axons of living neurons. Or they can be microprocessors. 

Image 3: AIDS-virus

Or the virus itself turns into a molecular qubit. If we are looking at the AIDS virus nothing denies that it could work as a qubit. The system requires a genome where data is loaded. Then the qubit virus will send electric signals to receivers that can be the living neuron. Or they can be microprocessors. 

Human brain neurotransmitters are molecular qubits. And molecular qubits can use for transmitting data between living neurons and non-organic microprocessors. There is a possibility that some viruses can transform into molecular qubits. DNA could be excellent data storage. And the outer core of the virus can send electric impulses to the receivers. 

The problem is how to make the internal data of the DNA transform electric signals. The answer can be simple. The retrovirus will infect the bacteria. And then that transforms the cell into a memory neuron. Or the DNA can turn the bacteria to be able to give electric shocks. Then the DNA would transfer its data by using those electric shocks as the transmitter. 

https://scitechdaily.com/quantum-breakthrough-scientists-extend-qubit-lifetimes/

https://scitechdaily.com/scientists-have-found-a-way-to-manipulate-digital-data-stored-in-dna/

https://en.wikipedia.org/wiki/Fullerene

Image: 

Image 1: https://scitechdaily.com/quantum-breakthrough-scientists-extend-qubit-lifetimes/

Image 2: https://en.wikipedia.org/wiki/Fullerene

Image 3: https://medizin.plus/krankheiten/hiv

https://designandinnovationtales.blogspot.com/

Could viruses be more active than researchers expected?

Images: 

(Left:) "Viruses are using information from their environment to “decide” when to sit tight inside their hosts and when to multiply and burst out, killing the host cell, according to a new scientific study. This illustration depicts a bacteriophage". (ScitechDaily.com/Shhh! Viruses Could Be Listening – And Watching!)

(Right:)"A delta bacteriophage. The first was identified in a new study in Frontiers in Microbiology to have binding sites for CtrA. A protein produced by the bacteriophage’s host that regulates the production of pili and flagella. The presence of these binding sites only in phages that require their host cells to have pili/flagella to infect them suggests that the phage is monitoring the presence of this protein to “decide” whether to stay put or replicate and emerge from its host cell. Credit: Tagide de Carvalho/UMBC" (ScitechDaily.com/Shhh! Viruses Could Be Listening – And Watching!)

Could viruses be more active than researchers expected? 

Some viruses might be more active than nobody expected. The bacteriophages can even swim to their target cell. So in that case virus will not just wait for the cell it swims to its target. This ability is suitable for bacteriophages that are hunting for bacteria from nature. 

There is the possibility that some viruses have a more complicated data-analyzation system than nobody expected. We know that the DNA plasmid of bacteria can operate as the chemical brain. But the new research shows, that also the DNA or RNA of viruses can act similar way, as the DNA plasmid of bacteria. 

So also, viruses might have this kind of chemical computer or chemical brain. The chemical brain of viruses could also have some kind of sensors. The proteins that form viruses could act as sensors and at least some viruses can swim to their target. 

The reason, why some people believe that especially bacteriophages must swim to their target is that those viruses must find the bacteria that are not resistant to those viruses. If viruses have this swimming ability they could revolutionize medical work. That ability makes it possible to create intelligent medicines that DNA or RNA controls. And this thing can make it possible to create also small but complicated nanomachines. 

When we look at the form of bacteriophages. It looks like a tiny space rocket. And the small feet of the virus can also help it to track the cells and swim to the target. The idea is that when a certain molecule hits a certain point of a virus. It activates an electrochemical reaction that moves the feet of the virus. And the form of the virus helps it adjust the direction where it moves. 

Then this genius system turns the virus upside down and makes it to "land" over the ion pump of the cell. The ion pump pulls the bottom of the virus to it. And then it simply pulls the genome of the virus to the cell. Or there is a possibility. That there is a cutter enzyme at the front of the DNA or RNA of the virus. When a virus touches the core of its victim it shoots its genome straight through the cell. 

https://scitechdaily.com/shhh-viruses-could-be-listening-and-watching/

Images: https://scitechdaily.com/shhh-viruses-could-be-listening-and-watching/

The electric pill and genome transfer can help the immune system to fight against viruses, including COVID-19.

Vaccines are playing a key role in virus infections. But the fact is that there is a new way that can boost the effectiveness of vaccines. The first one is to "teach" neurons that are communicating with immune cells. 

And the second would be to transfer the DNA sequences that are creating the immune defense from one person to another. In the second version, the DNA contains information on what kind of antigens the immune system needs to fight against some viruses that will transfer straight into the stem cells.

But then to electric shocks. Those electric systems will stimulate neurons that communicate with the immune system. There is a possibility that the recorded electric impulses of neural tracks of the virus patient will record. And then the system will use electric stimulation to teach similar neurons in another person's body. 

The electric shocks given to the right point of the ear can teach the neural system to activate the work of immune cells. The idea is that the electric stimulation stimulates nerves that are keeping contact with immune cells. The communication between the brain and the immune system is a well-known thing. And that communication can use to make the immune system more effective than without those electric shocks. 

Image 2) "Auricular Vagus Nerve Stimulation. Individualised auricular vagus nerve stimulation at the right time and with the right strength. Credit: Vienna University of Technology" (ScitechDaily.com/“Electric Pill” Shown To Help Patients With Severe COVID-19)

The electric pill stimulates neural tracks of nerves that control inflammatory reflexes. But don't leave your vaccine. The electric pill helps the immune system, but that immune reaction is too weak that it can defend the body against viruses. 

The electric stimulation can help the immune system because immune cells are interacting with neurons. So there is a possibility to use the neural tracks that have a responsibility, in communication with the immune system. 

As we know the best solution for immune defense is hybrid methods. The vaccine, along with neural stimulation can create the best possible results. But can we make vaccines even more effective?

Normal vaccines activate immune systems by helping immune cells detect the right antigen earlier. But the better version is to use genetic therapy where the DNA sequences that are storing genetic information of the viruses would transfer to the stem cells that are creating immune cells. 

In that version, the idea is that the immune cells can get information on how to fight against viruses from the reprogrammed immune cells. That kind of vaccine would make the immune system react earlier and more perfectly to the virus attack. The idea is that the hybrid methods are making immune systems more powerful than one single method. 

https://scitechdaily.com/scientists-show-transmission-of-epigenetic-memory-across-multiple-generations/

https://scitechdaily.com/electric-pill-shown-to-help-patients-with-severe-covid-19/

Image.1 (https://www.bioelectricsforhealth.com/post/what-are-viruses)

Image.2 (https://scitechdaily.com/electric-pill-shown-to-help-patients-with-severe-covid-19/)

Zombie cells and HIV viruses are promoting aging.

 

"Senescent cells, or “zombie cells,” are unique in that they ultimately cease multiplying but do not die off as expected." (ScitechDaily.com/Age Accelerating “Zombie Cells” – A New Study Sheds Light on These Unique Cells)

The term zombie cell means a cell that self-destruction is disturbed. That thing causes an effect, where those cells cannot die as they should. Those over-aged cells might affect aging and early stages of cancer. The forming of the zombie cell can be connected to the mitochondria. 

Mitochondria are small organelles that are giving energy to the cells. Somewhere in history mitochondria were independent organisms. Mitochondria have their DNA that is separated from the cell's main DNA.  And they divide independently.  When a cell dies the mitochondria should stop the energy production. 

But somehow the mitochondria continue their work after the calculated time. When the cell dies, the nucleus sends a mark to mitochondria to shut themselves down. When energy production of the cell ends it dies.

The metabolism products of the mitochondria are the mark that the cell is living. And even if the cell itself sends the mark that it's going to die, mitochondria continue their work that denies the immune cell to destroy that zombie cell.

Zombie cells are causing cancer because their DNA can face more damage than regular cells. And because those cells cannot die causes that those cells form "daughter cells", which DNA is not acting as it should. 

And that thing causes cancer. But the zombie cells can increase the aging process. The reason for that might be in the immune system. When the cell is aging it sends chemical marks that tell its condition to immune cells. When the cell is advancing in a certain stage in its life, it sends the "stand by for eating me" to immune cells whose mission is to remove remnants of the dead cell. Then the cell sends its final mark and the immune cell destroys it. 

That "stand by" mark tells that the core of the cell must turn thin. And the immune cells start to prepare for the destruction process. The purpose of that process is to deny the spread of programmed cell death. During that process, the antigens are affecting also the DNA by cutting its telomers. If the final mark of the death of the cells is not coming the immune cell continues its preparation process. 

And those antigens are starting to spread in the body. Those antigens are cutting the DNA in other cells. The thing that supports this theory is that HIV viruses are also increasing the speed of aging. The anti-HIV medicals are affecting the genetic material of the viruses. And also the immune system creates antibodies that affect the DNA and RNA molecule synthesis. 

Also, the virus itself copies itself to the DNA and that causes errors in the DNA structure. The HIV is the RNA-retrovirus. When the cell divides the RNA of the retrovirus can slip into the DNA. Because the cell makes a copy of the DNA through the RNA molecules. During that process, the cell copies the entire DNA to the daughter cell.

So the RNA molecules will travel above the DNA and transfer that code to the descendant cell. If there is the RNA of the HIV the copy process will be disturbed. That thing causes the descendant cell can have mutated cell organs. 

Because the DNA and RNA molecules are shorter, this means the base-pair order is disturbed or the DNA is damaged because of HIV that thing causes the aging of the cells to be faster than usual. And because the immune system and medicines must affect the DNA that thing causes damage to the DNA molecules all around the body.

https://scitechdaily.com/age-accelerating-zombie-cells-a-new-study-sheds-light-on-these-unique-cells/

https://scitechdaily.com/hiv-accelerates-aging-by-5-years/

Image: ) https://scitechdaily.com/age-accelerating-zombie-cells-a-new-study-sheds-light-on-these-unique-cells/

How was the COVID-19 Omikron variant formed?

 

 How was the COVID-19 Omikron variant formed?

The reason why the new COVID-19 variant formed was maybe the HIV patient in Africa. The reason why the COVID-19 Omikron variant is formed in Africa. Is that there must be the lowest vaccination level in the world. 

The fact is that some African nations even don't know how many citizens they have. And another thing is that how many people are visited, in COVID-19 tests in Africa? When we are talking about the COVID-19 infections we are talking about the confirmed infections. 

On this point, I must say that HIV infections are also possible in Europe and especially in the society of narcotics users and outcasted people. That means the COVID-19 can mutate also in Europe or Russia. 

And the thing is that the COVID-19 is not the only dangerous virus in the world. Also, other viruses can cause deadly infections. The mutation or ability to connect the new genomes to the DNA or RNA of the virus can transform any virus to dangerous. 

How to determine the term "confirmed infections?". In those cases, the confirmation should be made by using European standards. And those tests should be made by trained medical staff. So is those things reality in Africa? The thing is that the COVID-19 is creating new variants all the time. But the COVID-19 is not the only coronavirus that exists. The omikron variant can be formed from the other viruses. And the thing is that the genomes of the viruses are transforming all the time. While they are infecting some species or cell lines. 

The problem is that the things like HIV and other kinds of lost immune cells can cause that the vaccines are unable to work. If infected persons would not have immune cells the vaccines are unable to work. The loss of immune cells can cause by HIV. Or some medicines are making the immune system unable to work. The thing is that if some woman or man is hiding the HIV infection. 

That thing can cause the fast mutation of the virus. The problem with the countries in Africa is that there is no social security. And that causes that some prostitutes might hide their infection because that causes nobody will feed them. But also European tourists can get HIV or COVID-19 or both of them from Africa. And that thing means that the mutation can happen also in Europe. So the HIV patient is enough that the virus can infect the person. 

That means the COVID-19 is unpredictable. Many things are affecting the pandemics. And one of them is that if people are hiding that they are HIV-positive, that can cause the situation that the COVID-19 would infect the person. And that kind of infection can cause the mutation of the virus. This kind of infection can form new variants of viruses. And anyway, those variants can form also in Europe. Or anywhere on our planet.

https://scitechdaily.com/the-hunt-for-covid-variants-how-the-new-omicron-variant-was-found-and-what-we-know-so-far/

https://networkedinternet.blogspot.com/